Aphasia is primarily a disorder of:
Short, effortful speech with reduced phrase length
Left-hemisphere stroke in the MCA territory
Language processing affecting comprehension &/or expression
Memory and attention
Nonfluent aphasias are characterized by:
Normal prosody with paraphasias and reduced meaning
Short, effortful speech with reduced phrase length
Fluent but empty output
Normal prosody
The most common cause of aphasia is:
Cerebellar infarct
TBI
Left-hemisphere stroke in the MCA territory
Loss of motor plan
Fluent aphasias are characterized by:
Effortful output
Normal prosody with paraphasias and reduced meaning
Short, effortful speech with reduced phrase length
Agrammatism
Repetition is most severely impaired in:
Anomic aphasia
Conduction aphasia
Transcortical motor aphasia
Transcortical sensory aphasia
Paraphasias refer to:
Reduced attention
Made-up, meaningless words produced in fluent aphasia
Err. in word selection/ phoneme production in fluent speech
Difficulty producing phonemes
Neologisms are:
Made-up, meaningless words produced in fluent aphasia
Err. in word selection/ phoneme production in fluent speech
Real words with altered meaning
Words used in jargon aphasia only
A hallmark of Wernicke’s aphasia is:
Errors where sounds are substituted (“papple” for “apple”)
Good auditory comprehension
Nonfluent speech with relatively good comprehension
Poor auditory comprehension with fluent but empty speech
Broca’s aphasia is typically caused by damage to:
Posterior superior temporal gyrus
Left inferior parietal lobe
Left inferior frontal gyrus
Angular gyrus
A key characteristic of Broca’s aphasia is:
Errors where sounds are substituted (“papple” for “apple”)
Nonfluent speech with relatively good comprehension
Poor auditory comprehension
Severe jargon
Literal/phonemic paraphasias are:
Telegraphic speech
Errors where sounds are substituted (“papple” for “apple”)
Semantic substitutions
Neologisms
Repetition in Broca’s aphasia is:
Intact
Superior to spontaneous speech
Mildly impaired
Severely impaired
Wernicke’s aphasia is caused by damage to the:
Supplementary motor area
Superior longitudinal fasciculus
Posterior superior temporal gyrus
Insula
A hallmark of Wernicke’s aphasia is:
Nonfluent speech with relatively good comprehension
Strong repetition
Semantic substitutions
Poor awareness of deficits (anosognosia)
Global aphasia involves:
Severe jargon
Poor awareness of deficits (anosognosia)
Poor auditory comprehension
Severe impairments in all language modalities
The lesion for global aphasia typically involves:
Cerebellum
The entire left perisylvian region
Occipital cortex
Posterior superior temporal gyrus
