A key characteristic of Broca’s aphasia is:
Nonfluent speech with relatively good comprehension
Severe jargon
Poor auditory comprehension
Errors where sounds are substituted (“papple” for “apple”)
The most common cause of aphasia is:
Cerebellar infarct
Loss of motor plan
Left-hemisphere stroke in the MCA territory
TBI
Aphasia is primarily a disorder of:
Left-hemisphere stroke in the MCA territory
Memory and attention
Short, effortful speech with reduced phrase length
Language processing affecting comprehension &/or expression
A patient whose auditory comprehension is strong but who produces only a few words with great effort likely demonstrates:
Broca’s aphasia
Anomic aphasia
Conduction aphasia
Wernicke’s aphasia
A hallmark of Wernicke’s aphasia is:
Poor auditory comprehension with fluent but empty speech
Good auditory comprehension
Errors where sounds are substituted (“papple” for “apple”)
Nonfluent speech with relatively good comprehension
Transcortical mixed aphasia is characterized by:
Severe deficits in expression and comp. w/ preserved repet.
Intact repetition with poor auditory comprehension
Good reading comprehension
Fluent speech with prominent word-finding difficulty
Wernicke’s aphasia is caused by damage to the:
Insula
Superior longitudinal fasciculus
Supplementary motor area
Posterior superior temporal gyrus
Which feature BEST differentiates transcortical motor aphasia (TMA) from Broca’s aphasia?
Agrammatic spoken language
Intact repetition despite reduced initiation of speech
Nonfluent, effortful output
Poor auditory comprehension
A patient produces fluent but meaningless sentences filled with neologisms. This is:
Agrammatism
Jargon aphasia
Logorrhea
Hypophonia
A hallmark feature that differentiates Wernicke’s aphasia from anomic aphasia is:
Intact repetition
Fluent speech
Word-finding difficulty
Severely impaired auditory comprehension
A patient repeats perfectly but cannot initiate conversation. This suggests:
Conduction aphasia
Broca’s aphasia
Global aphasia
Transcortical motor aphasia
Conduction aphasia is linked to damage in:
The entire left perisylvian region
Right frontal lobe
Arcuate fasciculus
Precentral gyrus
Fluent aphasias are characterized by:
Short, effortful speech with reduced phrase length
Normal prosody with paraphasias and reduced meaning
Effortful output
Agrammatism
Frequent circumlocutions with relatively preserved grammar and comprehension indicate:
Broca’s aphasia
Anomic aphasia
Wernicke’s aphasia
Global aphasia
A hallmark of Wernicke’s aphasia is:
Nonfluent speech with relatively good comprehension
Semantic substitutions
Poor awareness of deficits (anosognosia)
Strong repetition
A patient with strong auditory comprehension but very limited output and severe agrammatism likely has:
Wernicke’s aphasia
Broca’s aphasia
Transcortical sensory aphasia (TSA)
Anomic Aphasia
Repetition in Broca’s aphasia is:
Superior to spontaneous speech
Mildly impaired
Intact
Severely impaired
Broca’s aphasia is typically caused by damage to:
Angular gyrus
Posterior superior temporal gyrus
Left inferior parietal lobe
Left inferior frontal gyrus
A patient shows minimal spontaneous speech, preserved repetition, and intact naming when prompted. This is characteristic of:
Transcortical motor aphasia
Wernicke’s aphasia
Mixed transcortical aphasia
Global aphasia
Perseveration is MOST common in:
Wernicke’s aphasia
Global aphasia
Broca’s aphasia
Conduction aphasia
Global aphasia is MOST accurately described as:
Severe expressive deficits with intact comprehension
Severe impairments in expr., comp., naming, reading, & rep.
Fluent speech with poor auditory comprehension
Normal prosody with paraphasias and reduced meaning
A patient shows echolalia, poor comprehension, and intact repetition. This pattern is typical of:
Transcortical Sensory Aphasia (TSA)
Anomic Aphasia
Conduction Aphasia
Transcortical Mixed Aphasia
In anomic aphasia, repetition is typically:
Absent
Poor
Severely impaired
Relatively intact
Global aphasia involves:
Severe jargon
Severe impairments in all language modalities
Poor awareness of deficits (anosognosia)
Poor auditory comprehension
Anomic aphasia is marked by:
Severe repetition impairment
Fluent speech with prominent word-finding difficulty
Nonfluent output
Severe deficits in expression and comp. w/ preserved repet.
The lesion for global aphasia typically involves:
The entire left perisylvian region
Posterior superior temporal gyrus
Occipital cortex
Cerebellum
Repetition is most severely impaired in:
Transcortical sensory aphasia
Anomic aphasia
Transcortical motor aphasia
Conduction aphasia
Mixed transcortical aphasia is characterized by which pattern?
Poor repetition, poor comprehension, poor naming
Severe deficits in expr. & comp. with preserved repetition
Strong naming with mild fluency deficits
Fluent output but good comprehension
Global aphasia is most commonly associated with a lesion involving:
Posterior cerebral artery infarct
Entire L perisylvian region, often from a large MCA stroke
Right temporal lobe
The cerebellum
Nonfluent aphasias are characterized by:
Normal prosody with paraphasias and reduced meaning
Normal prosody
Fluent but empty output
Short, effortful speech with reduced phrase length
A patient produces minimal spontaneous speech, has very poor comprehension, but can repeat long sentences verbatim. This most strongly suggests:
Transcortical sensory aphasia
Global aphasia
Wernicke’s aphasia
Mixed transcortical aphasia
Literal/phonemic paraphasias are:
Neologisms
Errors where sounds are substituted (“papple” for “apple”)
Semantic substitutions
Telegraphic speech
Transcortical Sensory Aphasia (TSA) is often mistaken for:
Anomic aphasia
Conduction aphasia
Broca’s aphasia
Wernicke’s aphasia with preserved repetition
Good repetition + good comprehension + word-finding difficulties =
Global aphasia
Anomic aphasia
Wernicke’s aphasia
Broca’s aphasia
A patient presents with severe word-finding problems, intact comprehension, normal repetition, and difficulty producing low-frequency nouns. This most likely reflects:
Broca’s aphasia
Conduction aphasia
Global aphasia
Anomic aphasia
The main linguistic hallmark of conduction aphasia is:
Poor auditory comprehension with fluent but empty speech
Nonfluent speech with relatively good comprehension
Severe impairments in all language modalities
Severely impaired repetition with good comprehension
A patient presents with echolalia, severely impaired comprehension, but intact repetition. This pattern best indicates:
Mixed transcortical aphasia
Global aphasia
Transcortical motor aphasia
Transcortical sensory aphasia (TSA)
A repetition vs. comprehension dissociation best distinguishes:
Anomia vs. agnosia
Transcortical vs. non-transcortical aphasias
Apraxia vs. dysarthria
Fluent vs. nonfluent aphasia
Transcortical Motor Aphasia (TMA) typically presents with:
Severely impaired repetition with good comprehension
Good repetition with reduced initiation of speech
Mild naming deficits
Severely impaired comprehension with good repetition
Press of speech with poor comprehension and anosognosia indicates:
Transcortical sensory aphasia
Broca’s
Transcortical motor aphasia
Wernicke’s aphasia
A patient can name objects and comprehend well, but pauses often due to difficulty retrieving specific nouns in conversation. This strongly suggests:
Wernicke’s aphasia
Mixed transcortical aphasia
Broca’s aphasia
Anomic aphasia
A patient with fluent speech, frequent phonemic paraphasias, poor repetition, and good comprehension most likely has:
Transcortical motor aphasia
Conduction aphasia
Transcortical sensory aphasia (TSA)
Global aphasia
A patient shows severely impaired naming, relatively fluent output, intact repetition, and poor auditory comprehension. This profile MOST strongly suggests:
Anomic aphasia
Transcortical sensory aphasia (TSA)
Wernicke’s aphasia
Conduction aphasia
The presence of logorrhea (press of speech) strongly differentiates:
Wernicke’s aphasia from conduction aphasia
Global from transcortical mixed aphasia
Wernicke’s from sensory aphasia
Broca’s from Transcortical motor aphasia
Paraphasias refer to:
Err. in word selection/ phoneme production in fluent speech
Made-up, meaningless words produced in fluent aphasia
Difficulty producing phonemes
Reduced attention
A patient with fluent output, impaired comprehension, intact repetition, and semantic paraphasias likely has:
Transcortical sensory aphasia (TSA)
Anomic aphasia
Broca’s aphasia
Global aphasia
A person with fluent speech, poor comprehension, and good repetition most likely has:
Conduction aphasia
Transcortical sensory aphasia
Wernicke’s aphasia
Mixed transcortical aphasia
Neologisms are:
Err. in word selection/ phoneme production in fluent speech
Words used in jargon aphasia only
Real words with altered meaning
Made-up, meaningless words produced in fluent aphasia