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Aphasia types and characteristics
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Global aphasia is most commonly associated with a lesion involving:
Entire L perisylvian region, often from a large MCA stroke
Right temporal lobe
Posterior cerebral artery infarct
The cerebellum
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Global aphasia is MOST accurately described as:
Severe impairments in expr., comp., naming, reading, & rep.
Fluent speech with poor auditory comprehension
Severe expressive deficits with intact comprehension
Normal prosody with paraphasias and reduced meaning
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A patient produces minimal spontaneous speech, has very poor comprehension, but can repeat long sentences verbatim. This most strongly suggests:
Mixed transcortical aphasia
Transcortical sensory aphasia
Global aphasia
Wernicke’s aphasia
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Mixed transcortical aphasia is characterized by which pattern?
Severe deficits in expr. & comp. with preserved repetition
Strong naming with mild fluency deficits
Poor repetition, poor comprehension, poor naming
Fluent output but good comprehension
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Which feature BEST differentiates transcortical motor aphasia (TMA) from Broca’s aphasia?
Intact repetition despite reduced initiation of speech
Nonfluent, effortful output
Agrammatic spoken language
Poor auditory comprehension
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A patient presents with severe word-finding problems, intact comprehension, normal repetition, and difficulty producing low-frequency nouns. This most likely reflects:
Anomic aphasia
Global aphasia
Broca’s aphasia
Conduction aphasia
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A patient shows minimal spontaneous speech, preserved repetition, and intact naming when prompted. This is characteristic of:
Transcortical motor aphasia
Global aphasia
Mixed transcortical aphasia
Wernicke’s aphasia
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The presence of logorrhea (press of speech) strongly differentiates:
Wernicke’s aphasia from conduction aphasia
Broca’s from Transcortical motor aphasia
Wernicke’s from sensory aphasia
Global from transcortical mixed aphasia
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Frequent circumlocutions with relatively preserved grammar and comprehension indicate:
Anomic aphasia
Broca’s aphasia
Global aphasia
Wernicke’s aphasia
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A patient with fluent output, impaired comprehension, intact repetition, and semantic paraphasias likely has:
Transcortical sensory aphasia (TSA)
Anomic aphasia
Broca’s aphasia
Global aphasia
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A patient can name objects and comprehend well, but pauses often due to difficulty retrieving specific nouns in conversation. This strongly suggests:
Anomic aphasia
Wernicke’s aphasia
Broca’s aphasia
Mixed transcortical aphasia
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A patient whose auditory comprehension is strong but who produces only a few words with great effort likely demonstrates:
Broca’s aphasia
Anomic aphasia
Wernicke’s aphasia
Conduction aphasia
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A patient with fluent speech, frequent phonemic paraphasias, poor repetition, and good comprehension most likely has:
Conduction aphasia
Global aphasia
Transcortical motor aphasia
Transcortical sensory aphasia (TSA)
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A patient presents with echolalia, severely impaired comprehension, but intact repetition. This pattern best indicates:
Transcortical sensory aphasia (TSA)
Global aphasia
Transcortical motor aphasia
Mixed transcortical aphasia
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A hallmark feature that differentiates Wernicke’s aphasia from anomic aphasia is:
Severely impaired auditory comprehension
Word-finding difficulty
Intact repetition
Fluent speech
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A patient shows severely impaired naming, relatively fluent output, intact repetition, and poor auditory comprehension. This profile MOST strongly suggests:
Transcortical sensory aphasia (TSA)
Anomic aphasia
Wernicke’s aphasia
Conduction aphasia
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Good repetition + good comprehension + word-finding difficulties =
Anomic aphasia
Broca’s aphasia
Global aphasia
Wernicke’s aphasia
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A patient produces fluent but meaningless sentences filled with neologisms. This is:
Jargon aphasia
Agrammatism
Logorrhea
Hypophonia
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Perseveration is MOST common in:
Global aphasia
Broca’s aphasia
Wernicke’s aphasia
Conduction aphasia
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A repetition vs. comprehension dissociation best distinguishes:
Transcortical vs. non-transcortical aphasias
Anomia vs. agnosia
Apraxia vs. dysarthria
Fluent vs. nonfluent aphasia
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A patient with strong auditory comprehension but very limited output and severe agrammatism likely has:
Broca’s aphasia
Wernicke’s aphasia
Anomic Aphasia
Transcortical sensory aphasia (TSA)
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Press of speech with poor comprehension and anosognosia indicates:
Wernicke’s aphasia
Broca’s
Transcortical motor aphasia
Transcortical sensory aphasia
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A patient shows echolalia, poor comprehension, and intact repetition. This pattern is typical of:
Transcortical Sensory Aphasia (TSA)
Transcortical Mixed Aphasia
Anomic Aphasia
Conduction Aphasia
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A patient repeats perfectly but cannot initiate conversation. This suggests:
Transcortical motor aphasia
Global aphasia
Conduction aphasia
Broca’s aphasia
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A person with fluent speech, poor comprehension, and good repetition most likely has:
Transcortical sensory aphasia
Mixed transcortical aphasia
Wernicke’s aphasia
Conduction aphasia
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25
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In anomic aphasia, repetition is typically:
Relatively intact
Poor
Severely impaired
Absent
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Anomic aphasia is marked by:
Fluent speech with prominent word-finding difficulty
Severe deficits in expression and comp. w/ preserved repet.
Nonfluent output
Severe repetition impairment
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Transcortical mixed aphasia is characterized by:
Severe deficits in expression and comp. w/ preserved repet.
Good reading comprehension
Fluent speech with prominent word-finding difficulty
Intact repetition with poor auditory comprehension
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Transcortical Sensory Aphasia (TSA) is often mistaken for:
Wernicke’s aphasia with preserved repetition
Conduction aphasia
Broca’s aphasia
Anomic aphasia
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Transcortical Motor Aphasia (TMA) typically presents with:
Good repetition with reduced initiation of speech
Severely impaired repetition with good comprehension
Severely impaired comprehension with good repetition
Mild naming deficits
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The main linguistic hallmark of conduction aphasia is:
Severely impaired repetition with good comprehension
Severe impairments in all language modalities
Nonfluent speech with relatively good comprehension
Poor auditory comprehension with fluent but empty speech
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Conduction aphasia is linked to damage in:
Arcuate fasciculus
Precentral gyrus
Right frontal lobe
The entire left perisylvian region
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The lesion for global aphasia typically involves:
The entire left perisylvian region
Posterior superior temporal gyrus
Occipital cortex
Cerebellum
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Global aphasia involves:
Severe impairments in all language modalities
Poor awareness of deficits (anosognosia)
Severe jargon
Poor auditory comprehension
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A hallmark of Wernicke’s aphasia is:
Poor awareness of deficits (anosognosia)
Strong repetition
Nonfluent speech with relatively good comprehension
Semantic substitutions
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Wernicke’s aphasia is caused by damage to the:
Posterior superior temporal gyrus
Supplementary motor area
Superior longitudinal fasciculus
Insula
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Repetition in Broca’s aphasia is:
Severely impaired
Superior to spontaneous speech
Intact
Mildly impaired
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Literal/phonemic paraphasias are:
Errors where sounds are substituted (“papple” for “apple”)
Telegraphic speech
Semantic substitutions
Neologisms
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A key characteristic of Broca’s aphasia is:
Nonfluent speech with relatively good comprehension
Poor auditory comprehension
Severe jargon
Errors where sounds are substituted (“papple” for “apple”)
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Broca’s aphasia is typically caused by damage to:
Left inferior frontal gyrus
Left inferior parietal lobe
Angular gyrus
Posterior superior temporal gyrus
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A hallmark of Wernicke’s aphasia is:
Poor auditory comprehension with fluent but empty speech
Good auditory comprehension
Nonfluent speech with relatively good comprehension
Errors where sounds are substituted (“papple” for “apple”)
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25
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Neologisms are:
Made-up, meaningless words produced in fluent aphasia
Words used in jargon aphasia only
Real words with altered meaning
Err. in word selection/ phoneme production in fluent speech
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Paraphasias refer to:
Err. in word selection/ phoneme production in fluent speech
Difficulty producing phonemes
Reduced attention
Made-up, meaningless words produced in fluent aphasia
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Repetition is most severely impaired in:
Conduction aphasia
Anomic aphasia
Transcortical sensory aphasia
Transcortical motor aphasia
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Fluent aphasias are characterized by:
Normal prosody with paraphasias and reduced meaning
Agrammatism
Short, effortful speech with reduced phrase length
Effortful output
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The most common cause of aphasia is:
Left-hemisphere stroke in the MCA territory
Cerebellar infarct
TBI
Loss of motor plan
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Nonfluent aphasias are characterized by:
Short, effortful speech with reduced phrase length
Fluent but empty output
Normal prosody
Normal prosody with paraphasias and reduced meaning
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Aphasia is primarily a disorder of:
Language processing affecting comprehension &/or expression
Memory and attention
Left-hemisphere stroke in the MCA territory
Short, effortful speech with reduced phrase length
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