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Anatomy and physiology of swallowing

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  • A reduced anterior–posterior tongue movement affects which stage?
    None
    Oral transit
    Esophageal
    Pharyngeal
  • The primary function of the arytenoids during swallowing is to:
    Elevate the velum
    Move anteriorly & medially to close the larynx
    Control saliva production
    Increase tongue pressure
  • Which cranial nerve provides sensory innervation to the supraglottic larynx?
    CN X vagus
    CN VII facial
    CN XII hypoglossal
    CN IX glossopharyngeal
  • Damage to the mandibular branch of CN V (trigeminal) would MOST likely cause difficulty with:
    Taste sensation
    Vocal fold adduction
    Jaw elevation during mastication
    Lip rounding
  • Which CN provides motor input to the palatoglossus muscle?
    CN Xll (hypoglossal)
    CN X (vagus)
    CN V (trigeminal)
    CN lX (glossopharyngeal)
  • Injury to the recurrent laryngeal nerve primarily disrupts:
    Motor innervation to the true vocal folds
    Sensory detection at the aryepiglottic folds
    Gag reflex and swallow trigger responsiveness
    Taste on the anterior tongue
  • Primary peristalsis occurs when:
    The bolus enters the esophagus
    The tongue elevates
    Upper Esophageal Sphincter contracts
    The pharynx squeezes
  • A lesion affecting CN VII (facial) would MOST likely result in:
    Posterior tongue numbness
    Weak jaw closure
    Absent cough reflex
    Reduced labial seal causing anterior spillage
  • Tongue elevation against the hard palate is primarily achieved by the:
    Styloglossus
    Palatoglossus
    Hyoglossus
    Genioglossus
  • The esophageal phase is controlled primarily by:
    Autonomic nervous system
    Spinal accessory nerve
    Cranial nerve XII (Hypoglossal)
    Spinal accessory nerve
  • The Upper Esophageal Sphincter opens as a result of:
    Pressure from the lungs
    Cricopharyngeal contraction
    Vocal fold adduction
    Hyolaryngeal traction & relaxation of the cricopharyngeus
  • Which structure prevents anterior spillage of the bolus?
    Uvula
    Epiglottis
    Lips & labial musculature
    Vagus reflex pathways
  • CN X (Vagus) is responsible for:
    Sensory to posterior 1/3 of tongue & pharynx
    Laryngeal sensation and motor closure
    Airway protection
    Facial expression
  • Reduced pharyngeal constriction leads to:
    Delayed swallow trigger
    Pyriform sinus residue
    Vocal fold paralysis
    Nasal emission
  • Loss of sensory input from the pyriform sinuses is MOST associated with risk of:
    Nasal regurgitation
    Reduced chewing strength
    Delayed airway protective responses
    Slowed AP transit
  • The lower esophageal sphincter function protects against:
    Aspiration
    Gastroesophageal reflux into the pharynx
    Nasal regurgitation
    Reflux into the esophagus
  • Reduced lingual lateralization would MOST affect:
    Bolus formation
    Upper Esophageal Sphincter opening
    Laryngeal closure
    Epiglottic inversion
  • Damage to CN XI (spinal accessory nerve) affects swallowing by:
    Impairing laryngeal elevation
    Decreasing Upper Esophageal Sphincter relaxation
    Redu. palatal elevation due to its fibers joining CN X (Vagu
    Weakening the pharyngeal constrictors
  • The oral transit stage ends when:
    Bolus enters valleculae
    Tongue base meets posterior pharyngeal wall
    Hyoid elevates
    Velum closes
  • Velopharyngeal closure prevents:
    Aspiration
    Globus sensation
    Cricopharyngeal spasm
    Nasal regurgitation
  • Loss of sensation from the posterior 1/3 of the tongue would MOST impact:
    Gag reflex & swallow trigger responsiveness
    Sensory detection at the aryepiglottic folds
    Reduced labial seal causing anterior spillage
    Motor innervation to the true vocal fold
  • Which muscle elevates the pharynx during swallowing?
    Inferior constrictor
    Stylopharyngeus
    Middle constrictor
    Superior constrictor
  • The laryngeal vestibule closes completely when:
    Epiglottis retroflexes
    Arytenoids contact the base of the epiglottis
    Upper Esophageal Sphincter opens
    Velum elevates
  • Which CN innervates the majority of pharyngeal constrictors?
    CN VII
    CN V
    CN IX & X
    CN XII
  • Which CN provides sensory input to the oropharynx to trigger the swallow?
    CN XII (Hypoglossal)
    CN VII (Facial)
    CN X (Vagus)
    CN IX (Glossopharyngeal)
  • Delayed pharyngeal swallow MOST commonly results in:
    Premature spillage into the airway
    Reduced Upper Esophageal Sphincter tone
    Nasal regurgitation
    Esophageal reflux
  • The pharyngeal swallow is primarily triggered when the bolus reaches the:
    Buccinator muscle
    Palatine tonsil
    Faucial pillars
    Velum
  • Mastication is MOST dependent on which cranial nerve?
    CN VII
    CN Xll
    CN lX
    CN V
  • Buccinator function during swallowing is to:
    Elevate the larynx
    Prevent pocketing in the lateral sulci
    Propel the bolus through the Upper Esophageal Sphincter
    Close the velopharyngeal port
  • CN VII (facial) contributes to swallowing by:
    Sensory to posterior 1/3 of tongue & pharynx
    Elevating velum
    Taste to anterior 2/3 of tongue & lip seal
    Triggering upper esophageal sphincter opening
  • CN IX (glossopharyngeal) provides:
    Motor to arytenoids
    Taste to anterior 2/3 of tongue & lip seal
    Sensory to posterior 1/3 of tongue & pharynx
    Motor innervation to the tongue
  • Hyolaryngeal elevation contributes MOST to:
    Velopharyngeal seal
    Nasal airflow
    Airway closure & Upper Esophageal Sphincter opening
    Tongue base retraction
  • Taste to the epiglottis is supplied by which cranial nerve?
    CN lX (Glossopharyngeal)
    CN Vll (facial)
    CN V (trigeminal)
    CN X (vagus)
  • Reduced relaxation of the Upper Esophageal Sphincter results in:
    Penetration only
    Pyriform sinus residue
    Nasal reflux
    Vallecular residue
  • Laryngeal closure during swallowing proceeds in what order?
    Epiglottis → arytenoids → true vocal folds
    Epiglottis → arytenoids → true vocal folds
    True folds → arytenoids → epiglottic tilt
    True vocal folds → false vocal folds → laryngeal vestibule
  • The oral preparatory stage is under control of:
    Voluntary cortical control
    Cerebellum only
    Brainstem only
    Vagus reflex pathways
  • Epiglottic inversion occurs primarily due to:
    Contraction of the epiglottic muscle
    Tongue base retraction & laryngeal elevation
    Preventing pocketing in the lateral sulci
    Airway closure & Upper Esophageal Sphincter opening
  • Reduced laryngeal sensation increases risk for:
    Nasal regurgitation
    Anterior spillage
    Penetration & silent aspiration
    Oral residue only
  • Tongue base retraction generates:
    Epiglottic closure only
    Shear pressure
    Cricopharyngeal hypertonicity
    Pharyngeal stripping wave
  • The muscle forming the primary structure of the Upper Esophageal Sphincter is the:
    Aryepiglottic muscle
    Cricopharyngeus
    Stylopharyngeus
    Thyroepiglottic muscle
  • The primary muscle responsible for lip closure during swallowing is:
    Buccinator
    Palatoglossus
    Orbicularis oris
    Masseter
  • Esophageal motility disorders primarily impact:
    Safety of the swallow
    Velopharyngeal function
    Lingual pressure
    Efficiency of bolus transit
  • Sensory loss to the valleculae would MOST directly impair:
    True vocal fold adduction
    Velopharyngeal closure
    Esophageal peristalsis
    Timely initiation of the pharyngeal swallow
  • The muscle that elevates and retracts the velum during oral transit is:
    Lips & labial musculature
    Palatopharyngeus
    Palatoglossus
    Levator veli palatini
  • CN XII (hypoglossal) damage most affects:
    Cricopharyngeal function
    Cough strength
    Lingual propulsion & bolus control
    Airway protection