Sometimes these drugs are referred to as sympatholytic because they opposes the downstream effects of organs innervated by the sympathetic nervous system.

blocks angiotensinII receptors prevent angiotensin II from activating the following activities and thus lower blood pressure.

drugs that inhibit angiotensin-converting enzyme which converts angiotensin I to angiotensin II. when angiotensin I is not converted to angiotensin II,  blood pressure is lowered

block the release of norepinephrine from sympathetic endings; lowers blood pressure by preventing this vasoconstrictor effect.

drugs that inhibit the release of ADH by the posterior pituitary which will decrease absorption of water at the kidneys decreasing blood volume and thus BP.

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These drugs block the effect of sympathetic nerves to cause vasoconstriction of blood vessels.

directly relaxing arteriolar smooth muscle by decreasing the phosphorylation of smooth muscle myosin light chain; this lowers arterial blood pressure by promoting arteriolar vasodilation and reducing TPN.

In the contractile cells, this type of medication primarily blocks L-type voltage gated calcium channels. TPR and BP are decreased as a result of reduced arteriolar smooth muscle contractile activity

Drugs that block  α1-adrenergic receptors; this results in vasodilation, which results in the  lowering arterial blood pressure.

This type of drug blocks calcium channels reducing the entry of calcium into myocardial cells and smooth muscle cells of arteriole blood vessel walls. This results in lower BP.

receptors found on smooth muscle of the bronchioles. Norepinephrine and epinephrine can bind and cause bronchodilation.

receptors found on autorhythmic cells of the heart such as the SA node and contractile myocardiocytes. Norepinephrine and epinephrine can bind and cause an increase in heart rate and contractile strength