receptors found on smooth muscle of arterioles of the coronary arteries and skeletal muscle. Norepinephrine and epinephrine can bind and cause vasodilation.

block the release of norepinephrine from sympathetic endings; lowers blood pressure by preventing this vasoconstrictor effect.

drugs that inhibit the release of ADH by the posterior pituitary which will decrease absorption of water at the kidneys decreasing blood volume and thus BP.

This type of drug blocks calcium channels reducing the entry of calcium into myocardial cells and smooth muscle cells of arteriole blood vessel walls. This results in lower BP.

drug that inhibits the body’s thirst center within the hypothalamus which causes a person to drink more -increased fluid intake causes an increase in blood volume and BP.

Grapefruit juice is contraindicated when taking this type of medication because it can increase the amount of medicine that enters your blood stream. As a result, your blood pressure can drop very suddenly.

In the contractile cells, this type of medication primarily blocks L-type voltage gated calcium channels. TPR and BP are decreased as a result of reduced arteriolar smooth muscle contractile activity

Sometimes these drugs are referred to as sympatholytic because they opposes the downstream effects of organs innervated by the sympathetic nervous system.

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Drugs that block β1 receptors decrease heart rate and force of contraction resulting in a reduction of cardiac output and blood pressure.

These drugs block the effect of sympathetic nerves to cause vasoconstriction of blood vessels.

receptors found on autorhythmic cells of the heart such as the SA node and contractile myocardiocytes. Norepinephrine and epinephrine can bind and cause an increase in heart rate and contractile strength

Drugs that act on the medulla oblongata to reduce sympathetic output; lowers B/P by preventing the effect of sympathetic activity and promoting arteriolar vasoconstriction.