*presence, location and size of vegetation. *valve dysfunction. *valve dysfun impact on LV size and systolic func. *other comp (PE...)
Oops!
Check
Okay!
Check
15
IE results from _______ or _______ infection of the endocardial surface of the heart, including valves.
bacterial or fungal
Oops!
Check
Okay!
Check
15
what is the treatment for IE and for how long and prevention.
Vancomyocin or ampicillin for 4-6weeks of high dose IV antibiotic therapy.
after complete therapy, pt should be educated on dental hygiene, prophylaxis need.
Oops!
Check
Okay!
Check
15
Infective Endo is defined as a localized infection anywhere on the _________.
Endocardium.
Oops!
Check
Okay!
Check
15
Echo Parameter consistent with risk vegetation size
a) <7mm = _____% complication
b) >11mm = _____% complication
a) <10%
b) >50%
Oops!
Check
Okay!
Check
15
what are some IE complications.
*embolization (left: stroke, ischemia, renal failure, right: PE or pneumonia)
* Leaflet perforation, flail * acute regurge *tamponade *HF
Oops!
Check
Okay!
Check
15
How do we diagnose IE. explain steps
Dukes Criteria: Clinical, Labs and echo findings
Oops!
Check
Okay!
Check
15
these two imaging modalities show promise for the evaluation for IE
MRI and CT
Oops!
Check
Okay!
Check
thief
Give points!
5
10
15
20
25
rocket
Go to first place!
Okay!
shark
Other team loses 15 points!
Okay!
thief
Give points!
5
10
15
20
25
15
What are the Risk Factors for IE
*Prosthetic Valve *recent Hx of invasive procedure (25%) *IV abuse
*structural or congenital heart disease
Oops!
Check
Okay!
Check
15
____ valve has 3 times more involvement in an emoblic event.
Mitral valve
Oops!
Check
Okay!
Check
15
________ is associated with significant morbidity and mortality (20-25%) and if left untreated 100% mortality rate.
IE
Oops!
Check
Okay!
Check
15
Pathogenesis of IE. (what needs to happen for IE to occur)
1. Endocardial surface injury 2. Platelet-fibrin-thrombus formation at site of injury
3. Bacterial entry in circulation 4. bacterial adherence to wall
Oops!
Check
Okay!
Check
15
What are clinial ways to identify IE?
Clinical course (acute or subacute)
Host substrate (native valve)
Specific infecting Microorganism