Unit 6A Sigs | Baamboozle - Baamboozle | The Most Fun Classroom Games!

Distinguish between cardiac conduction blocks (sinus block; bundle branch block; hemiblock) on an EKG. (SIGS 6.1b)

A: Sinus block; B: LBBB; C: RBBB; D: Hemi/fascicular (Left posterior hemiblock is associated with a frontal plane QRS axis more positive than +120°)

Oops!

Check

Okay!

Check

Discuss the effects of alcohol on the heart. How can alcoholism lead to thiamine deficiency and exacerbate DCM? (SIGS 6.2b)

myocyte damage/necrosis, mitochon. degeneration, fibrosis, chamber dilation; deficiency d/t poor nutrition & poor vitamin absorption

Oops!

Check

Okay!

Check

What is the MOA of Colchicine and how does it relate to the management of pericarditis?(Sigs Case 6.2a)

used as a theoretical anti-inflamm. agent; inhibits IL-1 from cascading, interferes w/ NFK-B; binds to microtubular tubulin in neutrophils-> inhibits mitosis

Oops!

Check

Okay!

Check

Explain how atropine alters conduction. (SIGS 6.1b)

M2 muscarinic antagonist--> block muscarinic receptor--> decrease parasympathetic response--> increase HR, decrease phase 4 of nodal cells

Oops!

Check

Okay!

Check

heart

Other team wins 20 points!

Oops!

rocket

Go to first place!

Okay!

shark

Other team loses 25 points!

Okay!

baam

Lose 20 points!

Oops!

Explain the effects of electrolyte imbalance on cardiac conduction and the impact erythromycin would have. (SIGS 6.1a)

alter depol/ pol/ repol; macrolide antibiotic w/ AE prolonged QT intv. d/t blockage of K+ channels--> delays phase 3 repolariz.--> more intracellular K+--> EADs

Oops!

Check

Okay!

Check

baam

Lose 15 points!

Oops!

seesaw

Swap points!

Okay!

fairy

Take points!

thief

Give points!

Explain the mechanism of action of amiodarone and the rationale for its use in dysrhythmias. (SIGS 6.2b)

Class III anti-arryth; MOA: blocks VG K+ channels --> inhibit Phase 3 repol--> prolong cardiac action potential & refractoriness--> decr. SA node firing rate

Oops!

Check

Okay!

Check

Contrast normal systolic and diastolic function in a healthy heart with the systolic and diastolic function of HCM.(Sigs Case 6.3b)

may have average systolic funct. but poor diastolic funct.; diastolic dysf. will have preserved EF; systolic dysf will have reduced EF

Oops!

Check

Okay!

Check

Describe integrative approaches to atrial fibrillation. (SIGS 6.1b)

Med. diet, stress reduction, no alcohol or caffeine, diet high in magnesium, fish oil, CoQ10

Oops!

Check

Okay!

Check

Describe the role of INR monitoring in anticoagulation therapy and explain the need for bridge therapy with warfarin. (SIGS 6.1a)

monitor coagulability (maintain optimal level- not too low or too high); Bridge therapy for warfarin due to taking several days to achieve therapeutic effects

Oops!

Check

Okay!

Check

lifesaver

Give 10 points!

Oops!

seesaw

Swap points!

Okay!

rocket

Go to first place!

Okay!

banana

Go to last place!

Oops!

thief

Give points!

rocket

Go to first place!

Okay!

fairy

Take points!

thief

Give points!

Describe the mechanism(s) of action of diltiazem and its effect on cardiac conductivity (SIGS 6.1a)

MOA: inhibits inflow Ca2+ ions into cardiac smooth muscle (SM) during depolarization; Decr. intracellular Ca2+ --> incr. SM relaxation/vasodilation--> decr. BP

Oops!

Check

Okay!

Check

Describe the pathogenesis of coxsackievirus type B and how it relates to viral myocarditis. (Sigs Case 6.2a)

GI tract (stable at low gI pH)→infects mucosal epithelial cells→viremia→infects + lyse heart/pleural surfaces

Oops!

Check

Okay!

Check

Describe the alterations in vascular reactivity that result from endothelial damage. (SIGS 6.3a)

damage to endothelium--> dysfunction (inability to react to vasodilation)--> structural remodeling--> alters function

Oops!

Check

Okay!

Check

Restart Join for Free

Your experience on this site will be improved by allowing cookies.